Milandr píše:
Na regeneraciu RNG doporucuje Berksonov protokol : Silymarin 900 mg, ALA 600 mg a selen 400 mcg, mozne rozdelit na viac davok.....Spolahlivo to funguje doporucuje sa uzivat 21 dni,cize 3 tyzdne.
Piste, prosim, tyto prispevky pokud mozno do sekce kde se o jatrech diskutuje a ne do inzerce.
Kazdopadne kdyz uz se to tu rozebira, tak si dovolim reagovat.
ALA je opravdu velmi ucinna latka v mnoha ohledech. Osobne bych vsak takto vysoke davky ALA v zadnem pripade u nikoho nedoporucoval!
ALA je velmi ucinny mobilizator rtuti a ma obrovskou schopnost prostoupit mozkovou barierou (BBB). V takovem pripade je potreba zvysene opatrnosti s ALA u lidi, kteri maji toxicitu rtuti, v puse maji spoustu amalaganu, maji problemy s detoxikaci (temer vetsina nemocnych) ci maji porusenou prave mozkovou barieru (Leaky Brain Sydnrome). Takova osoba se muze vystavovat zvysenemu riziku poruchy funkce mozku. Na to poukazuje i Andy Cutler ci Amy Yasko.
Mnohem efektivnejsi je R-ALA, ktera jiz nepotrebuje enzymatickou reakci NADH, avsak i tak davkovat pouze v minimalnich davkach rozdelenych v prubehu dne a nasledne nekolik dni vynechat.
Dosti lidi v me praxi nema strevni trakt a jatra v poradku. Rozjet v takovem pripade ve velkem mobilizaci kovu, bez dostatecne funkcnich vsech vylucovacich organu vcetne ledvin a bez doporuceni mineralnich koupeli ci infrasaun, muze zpusobovat velmi neprijemne stavy. A nejedna se tady o zadny Herx.
Nerikam, ze tento protokol nefunguje, ale ve sve praxi pouzivam setrnejsi zpusoby podpory jater a to primarne u lidi s toxicitou tezkych kovu ci amalgany v puse. Primym zvysenim glutathionu, dale jeho recyklaci (vcetne velmi nizkych davek R-ALA), prekursory + zvyseni exkrece zluci. Podporou methylacnich a transulfuracnich cest. A naslednou eliminaci skrze strevni trakt pomoci thiolu, methylkremicitych gelu ci NDF, vlakniny a probiotickych bakterii.
Reference:
ALA given intravenously to rats at doses of 37.5-300 μM/kg was shown to increase inorganic mercury release in bile by 1,200-4,000 percent immediately after mercury exposure.67 Levels of released inorganic mercury remained at a 300-700 percent elevation, even three hours after dosing with ALA. If mercury was injected 24 hours prior to the administration of ALA, the increase in release of inorganic mercury was substantially less, but was still elevated 140-330 percent.
A lower dose of ALA (37.5 μM/kg) was more effective than higher doses at increasing the biliary elimination of methylmercury.There was disconcerting evidence from this study, however, that ALA may also alter the tissue distribution of mercury and other heavy metals.
Although levels of inorganic mercury and methylmercury in the kidney dropped significantly,
levels of inorganic mercury also increased significantly in the brain, lung, heart, and liver tissue.
Methylmercury levels had also increased in the brain, intestine and muscle of the rats given ALA. The same phenomenon occurred in rats exposed
to cadmium and given the same doses of ALA.
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